Rb family proteins enforce the homeostasis of quiescent hematopoietic stem cells by repressing Socs3 expression

نویسندگان

  • Eunsun Kim
  • Ying Cheng
  • Elisabeth Bolton-Gillespie
  • Xiongwei Cai
  • Connie Ma
  • Amy Tarangelo
  • Linh Le
  • Madhumita Jambhekar
  • Pichai Raman
  • Katharina E Hayer
  • Gerald Wertheim
  • Nancy A Speck
  • Wei Tong
  • Patrick Viatour
چکیده

Prolonged exit from quiescence by hematopoietic stem cells (HSCs) progressively impairs their homeostasis in the bone marrow through an unidentified mechanism. We show that Rb proteins, which are major enforcers of quiescence, maintain HSC homeostasis by positively regulating thrombopoietin (Tpo)-mediated Jak2 signaling. Rb family protein inactivation triggers the progressive E2f-mediated transactivation of Socs3, a potent inhibitor of Jak2 signaling, in cycling HSCs. Aberrant activation of Socs3 impairs Tpo signaling and leads to impaired HSC homeostasis. Therefore, Rb proteins act as a central hub of quiescence and homeostasis by coordinating the regulation of both cell cycle and Jak2 signaling in HSCs.

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عنوان ژورنال:

دوره 214  شماره 

صفحات  -

تاریخ انتشار 2017